Preliminary scientific studies suggest conolidine could inhibit distinct ion channels, minimizing neuronal excitability and limiting ache indicators. This mechanism is especially relevant in neuropathic pain, in which irregular signaling results in persistent discomfort. Furthermore, conolidine appears to affect G protein-coupled receptor (GPCR) pathways integral to suffering percepti... https://joseonb909gpc3.wikirecognition.com/user