Metabolic alterations affiliated with T2D are very well characterised by epidemiological and research based scientific studies. The pathogenesis and development of T2D is ascribed to four mechanisms; improved Superior glycation conclude products (AGE) development, improved polyol pathway flux, activation of protein kinase C (PKC) isoforms, and greater hexosamine pathway flux[seven]. https://www.directivepublications.org/journal-of-diabetology-research/